Volume 21, Issue 124 (10-2014)                   RJMS 2014, 21(124): 90-95 | Back to browse issues page

XML Persian Abstract Print


Download citation:
BibTeX | RIS | EndNote | Medlars | ProCite | Reference Manager | RefWorks
Send citation to:

Modarreszadeh M, Astaraki A. The pathophysiology and treatment of rhegmatogenous retinal detachment. RJMS 2014; 21 (124) :90-95
URL: http://rjms.iums.ac.ir/article-1-3376-en.html
Iran University of Medical Sciences
Abstract:   (5540 Views)
 

Background: The pathophysiology of rhegmatogenous retinal detachment (RRD) has remained a challenging subject in ophthalmology. The aim of this study was evaluating the leading causes and treatment options of RRD.

 

Methods: The study was performed by a review of 10 years (2004-2014) related literature emerged in PubMed on the pathophysiology and treatment of rhegmatogenous retinal detachment (RRD).

 

Results: Retinal attachment is mainly due to two factors, RPE pump and inter photoreceptor matrix (IPM). The crucial factor in the formation of RRD is posterior vitreous detachment (PVD), started by liquefaction then syneresis and completed by gel contracture. If the PVD causes a symptomatic tear, RD ensues in up to 90% of eyes. PVD may not be detectable preoperatively and it must be detected during the operation by using triamcinolone acetonide (TA), indocyanin green (ICG) or Brilliant Blue (BBG).

Conclusion: The main factor for developing RRD is PVD (traction). Essential treatments of RRD include traction neutralization (SB or pneumatic retinopexy) or elimination of traction using Pars plana vitrectomy (PPV). The less used method is Pneumatic retinopexy
Full-Text [PDF 636 kb]   (4563 Downloads)    
Type of Study: review article | Subject: Ophthalmology

Add your comments about this article : Your username or Email:
CAPTCHA

Rights and permissions
Creative Commons License This work is licensed under a Creative Commons Attribution-NonCommercial 4.0 International License.

© 2024 CC BY-NC-SA 4.0 | Razi Journal of Medical Sciences

Designed & Developed by : Yektaweb